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dc.contributor.authorNg'ang'a, Zipporah W.
dc.contributor.authorMaina, Alice N.
dc.contributor.authorKnobel, Darryn L.
dc.contributor.authorJiang, Ju
dc.contributor.authorHalliday, Jo
dc.contributor.authorFeikin, Daniel R.
dc.contributor.authorCleaveland, Sarah
dc.contributor.authorJunghae, Muthoni
dc.contributor.authorBreiman, Robert F.
dc.contributor.authorRichards, Allen L.
dc.contributor.authorNjenga, M. Kariuki
dc.date.accessioned2014-12-03T08:25:43Z
dc.date.available2014-12-03T08:25:43Z
dc.date.issued2012
dc.identifier.citationEmerging Infectious Diseases, Volume 18, Number 2—February 2012en_US
dc.identifier.issn1080-6059
dc.identifier.urihttp://wwwnc.cdc.gov/eid/article/18/2/11-1372_article#suggestedcitation
dc.identifier.urihttp://hdl.handle.net/123456789/300
dc.description.abstractTo determine previous exposure and incidence of rickettsial infections in western Kenya during 2007–2010, we conducted hospital-based surveillance. Antibodies against rickettsiae were detected in 57.4% of previously collected serum samples. In a 2008–2010 prospective study, Rickettsia felis DNA was 2.2× more likely to be detected in febrile than in afebrile persons.en_US
dc.language.isoenen_US
dc.publisherCenters for Disease Control and Preventionen_US
dc.titleRickettsia felis Infection in Febrile Patients, Western Kenya, 2007–2010en_US
dc.typeArticleen_US
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